• Discuss the anatomy of hip and anterior pelvis.
• Diagnose dysfunction and pathologies of the hip and anterior pelvic structures.
• Order appropriate imaging and clinically correlate findings with conditions affecting the hip and anterior pelvis conditions.

• Discuss meniscus anatomy with emphasis on collagen network.
• Identify how collagen network can predict the type of tear and imaging manifestations.
• State how root ligaments are involved in injury, pain and OA.
• Correlate certain types of meniscal dysfunction to clinical presentation

Hour 1

• Select the appropriate imaging modality for suspected foreign bodies
• Identify foreign bodies on radiographs
• Identify foreign bodies CT
• Identify foreign bodies MRI
• Identify foreign bodies Ultrasound

Hour 2

• List various indications for radiographic evaluation in relationship to specific clinical scenarios
• List various limitations for radiographic evaluation in relationship to specific clinical scenarios
• Identify fracture patterns on radiographic imaging
• Identify radiographic abnormalities within the soft tissues

 Hour 3

• List various indications for MRI and CT imaging evaluation in relationship to specific clinical scenarios
• List various limitations for MRI and CT imaging evaluation in relationship to specific clinical scenarios
• Identify osseous landmarks on MRI and CT imaging
• Identify abnormalities within the bones and soft tissues on MRI and CT imaging

 Hour 4

• List various indications for ultrasound imaging evaluation in relationship to specific clinical scenarios
• List various limitations for ultrasound imaging evaluation in relationship to specific clinical scenarios
• Identify osseous landmarks on ultrasound imaging
• Identify abnormalities within the bones and soft tissues on ultrasound imaging

• Conduct and evidence-based assessment of the diagnostic, therapeutic and clinical utility of routine and repeat radiographs (without red flags) for the structural and functional assessment of the spine by chiropractors
• Summarize the current evidence for the use of spinal x-rays within chiropractic practice, with consideration to the related risks and benefits
• Determine if diagnostic imaging influences clinical outcomes in patients with low back pain presenting for chiropractic care
• Analyze whether an association between vertebral endplate structural defects and back pain exists
• Assess the relationship between movement-related pain in the shoulder and the number or severity of rotator cuff MRI findings

• Define HIPAA and identify fundamental components of the HIPAA law.
• Explain the concept of Protected Health Information and provide examples.
• Describe the key provisions of the HIPAA Privacy Rule, including patient rights and provider responsibilities.
• Summarize the HIPAA Security Rule and its requirements for safeguarding electronic Protected Health Information.
• Identify common HIPAA violations and their potential consequences.
• Explain the importance of Business Associate Agreements and their role in HIPAA compliance.
• Describe the steps involved in developing a HIPAA compliance plan.
• Discuss the impact of emerging technologies (e.g., telehealth, AI) on HIPAA compliance.

• List the major divisions and subcomponents of the neurological examination. 
• List, contrast and compare the neurological signs associated with “release phenomenon” and “deficit phenomena”.
• Contrast and compare the effects of neurological lesions on the neurological examination of the central nervous system versus the peripheral nervous system.
• Recite the implications of neurological examination findings on disorders involving various components of the central and peripheral nervous system.
• Draw the anatomy of the lower motor neuron in its relation to the spinal cord, nerve root, spinal nerve, and the neuro-myal junction.
• Describe the physical positioning of the patient during aspects of the neurological examination.
• List the subjects to question when in taking a good patient history.
• Describe and perform aspects of the neurological examination of the Cranial Nerves of the patient.
• Draw the neurologic pathways associated with the eye light reflexes.  Include reference to cells of the retina, the optic nerve, the optic chiasm, the optic tract, the Pretectal nucleus, and the Edinger Westphal nucleus.
• Describe and perform aspects of the neurological examination of the Motor Systems of the patient.
• Describe and perform aspects of the neurological examination of the Sensory Systems of the patient.
• Recite findings related to neurological “drift” performed during the neurological examination.
• Describe and perform aspects of the neurological examination of the Reflex Systems of the patient.
• Illustrate components of the muscle stretch reflex including the role of the Alpha motor neuron, Gama motor neuron, and large sensory fibers in relation to the muscle fibers and spinal cord.
• Discuss the common shorthand used for grading muscle stretch reflex responses and discuss the results of the neurological examination associated with each grade.
• Perform and discuss three versions of the extensor plantar response (extensor toe sign, Babinski sign).
• Describe and perform aspects of the neurological examination of movement of the patient.

Hour 1

• Compare the differences between acute and chronic inflammation.
• Associate background inflammation with chiropractic treatment outcomes.
• Discuss the aging trends in the U.S. population.
• Summarize the role of aging on background inflammation.
• Explain the differing outcomes of disc disease and radiculitis driven by inflammation.

Hour 2

• Identify the mechanisms by which macrophages activate inflammation.
• Discuss the historical trends in long chain fatty acid intake in relation to inflammation.
• Explain the dependency on the COX 2 enzyme both in inflammatory up-regulation and in resolution.
• Recognized dietary induced “meta-inflammation” and the dietary patterns that induce it.
• Summarize the role of phytonutrients in limiting dietary induced inflammation.

Hour 3

• Define immunosenescence and “inflammaging”.
• Summarize the role of autophagy in limiting inflammaging.
• Summarize the role of apoptosis in eliminating senescent immune cells.
• Examine the phytonutrients with senomorphic/pro-autophagy properties.
• Examine the phytonutrients with senolytic/pro-apoptotic properties.

Hour 4

• Describe the role of vagal nerve activation in regulating the transition to inflammatory resolution.
• Examine the splenic cholinergic anti-inflammatory pathway.
• Outline the mechanisms by which vagal activation increases specialized pro-resolving mediator (SPMs) production from omega-3 fatty acids.
• Summarize the role of transcutaneous vagal nerve stimulation (tVNS) in the inflammatory to pro-resolving transition.
• Contrast the inflammatory suppressing and pro-resolving treatment strategies.

• Explain the basic principles of nutrigenomics, focusing on the interaction between nutrition, genetics, and health outcomes, with an emphasis on conditions like headaches.
• Analyze how single nucleotide polymorphisms (SNPs) and other genetic variations impact nutrient metabolism and their potential role in triggering headaches, such as those linked to impaired methylation.
• Examine the mechanisms by which a lack of essential nutrients (e.g., magnesium, B vitamins) contributes to headaches, and how nutrigenomics can inform targeted nutritional interventions.
• Design tailored dietary strategies based on genetic testing results, addressing deficiencies or imbalances that may exacerbate headache conditions.
• Assess the benefits and limitations of using nutrigenomic testing to guide clinical decision-making for headache management and prevention, highlighting the importance of personalized nutrition in improving patient outcomes.

History and Background

• Recognize the historical emergence of spinal manipulation (SMT) throughout numerous time periods and among different professions
• Outline the development of the chiropractic profession from the late 1800s to modern day
• Briefly review existing and historical models of spinal manipulative therapy (SMT)

What’s that sound?

• Distinguish cavitation from tribonucleation and explore the relevance of the popping sound with SMT.

 Mechanisms

• Integrate the concept of mechanotransduction into your understanding and appraisal of SMT mechanisms
• Outline different ways methods to provide mechanical stimulation during chiropractic treatment
• Explain what the evidence tells us about joint motion, gapping and location of popping sounds with SMT
• Explore contemporary areas of scientific inquiry into the neurophysiologic mechanism(s) of action for high-velocity spinal manipulation
• Describe how SMT may influence pain modulation
• Critique the evidence relating to the influence of SMT on blood biomarkers and immunity
• Discuss the potential brain and sensorimotor functional impacts of SMT
• Appreciate the relevance of spinal stiffness and the potential impact of SMT on spinal biomechanics
• Summarize existing evidence relating to the SMT and muscle function.

Clinical Considerations

• Describe clinical factors that could influence how SMT exerts its effect(s).
• Discuss existing evidence on the dose-response relationship between SMT and clinical outcomes.
• Recognize the importance of contextual factors, patient expectations and comfort in driving positive clinical outcomes.
• Briefly summarize existing research on studies evaluating SMT for non-spinal conditions.

Course Conclusion

• Propose an integrated, conceptual model of how SMT may exert its effects for our patients.
• Recognize that chiropractic care helps patients via many aspects of the clinical encounter.

• Explain the origin of the vagus nerve, its anatomy, and its critical role in regulating bodily functions, including its connection to the sympathetic and parasympathetic nervous systems.
• Describe common causes of headaches and explore how the vagus nerve contributes to the pathophysiology of headache conditions.
• Discuss the science behind vagus nerve stimulation, its impact on neurological pathways, and its therapeutic benefits for treating headaches, epilepsy, treatment-resistant depression, reduced anxiety, and improved mood.
• Assess the efficacy and limitations of vagus nerve stimulation as a non-invasive treatment for headache reduction, referencing current research and clinical findings.
• Review the range of vagus nerve stimulation devices, from surgically implanted medical devices to over-the-counter options, and discuss their appropriate uses and considerations.

• This contains all courses with the educational objectives listed from Management of Common Conditions 269 through Management of Common Conditions 271

• Simplify the progressive cause-effect relationship of disease from biochemical imbalances to physiological disturbances to cellular/organ degeneration to disease manifestation
• Identify the five primary underlying etiologies of virtually all chronic degenerative disease
• Analyze the most common antecedents and triggers that predispose your patients to disease
• Devise an evaluation algorithm that efficiently guides your testing recommendations
• Discover novel in-office testing procedures that cost-effectively demonstrate patients’ nutrient insufficiencies; unmanaged stress; toxic burden; and sleep “debt”
• Evaluate Heart Rate Variability test results to easily measure and demonstrate patients’ stress adaptability as well as their response to treatment modalities
• Discriminate the meaningful information gleaned from Bio-Impedance Analysis (BIA) testing for monitoring your patients’ health restoration progress
• Interpret evaluation results to efficiently identify which primary factors are contributing to your patients’ health challenges
• Prioritize treatment plans that address both short-term goals (symptomatic relief) and long-term goals (correction, stabilization and prevention)
• Distinguish which nutraceuticals best support patient needs based on history, exam, testing and symptomatology
• Communicate the functional medicine model of health and disease with your patients with greater clarity and ease

• Properly document “doctor thinking” daily in routine patient visit documentation
• Recognize the role of the PART documentation process in Routine Office Visit notes
• Identify and execute the key components of written assessment in daily documentation
• Recognize aspects of documentation and coding of Route Office Visits (ROV) whether active treatment, preventative maintenance, or wellness care.
• Distinguish the unique components of Subjective, Objective, Assessment and Plan

• Apply the changes in the 2022 ICD-10 code set to the clinical diagnosis process
• Compare examination findings, couple with history, to select the most appropriate written diagnosis
• Document within the clinical record your thought process of selecting diagnostic codes
• Classify diagnoses in order of severity and hierarchy to match projected treatment plan

• Discuss the theories of why aging is now seen as a disease, characterized by chronic inflammation, which could potentially be stopped or even reversed. 
• Share how persistent, low-level inflammation (inflammaging) contributes to the deterioration of cellular and systemic functions, driving the aging process and the development of age-related chronic diseases.
• Examine the role of mitochondrial dysfunction in aging, exploring how improving mitochondrial health can promote longevity and reduce age-related decline.
• Identify clinical strategies to integrate mitochondrial health and anti-inflammatory practices into patient care to enhance healthy aging and mitigate the impact of chronic diseases.
• Introduction to low-level laser therapy, its core principles, and its mechanism of action for healing and optimizing cellular function.
• Explain the role of the mitochondria in cellular energy production and overall health.
• Review evidence-based applications of low-level laser therapy in promoting mitochondrial health via evidence-based research findings and data.
• Discuss practical approaches for integrating low-level laser therapy into clinical practice to enhance patient outcomes in managing mitochondrial dysfunction and promoting cellular rejuvenation.
• Share the global impact of metabolic dysfunction and discuss the effects of obesity driving chronic inflammation and its link to chronic health conditions.
• Discuss the mechanisms through which excess body fat and underlying metabolic dysfunction contributes to inflammatory processes and hormonal/endocrine dysfunction.
• Elaborate on clinical insights into effective interventions for combating obesity, improving metabolic health, and reducing inflammation, including dietary changes, physical activity, and sleep.
• Explore emerging therapies and technologies aimed at improving metabolic function and reducing inflammation, focusing on their potential applications in clinical practice.
• Discuss mechanisms of aging and how the aging process is being challenged by both conventional and alternative medical communities. 
• Provide a comprehensive analysis of nutritional, dietary, and lifestyle-based interventions that have shown promising outcomes for improving longevity and quality of life outcomes. 
• Overview of major concepts, themes, and discussions about the importance of diet, nutrition, and controlling blood sugar to optimize metabolic capacity to maximize aging. 
• Examine emerging research on cellular regeneration and mitochondrial function as critical components in slowing the aging process and enhancing longevity.

• Evaluation of post-partum patients
• Common complaints and assessment – LBP/SI, pelvic floor dysfunction, DRA
• Impact of birth type/healing time frame
• Biomechanics of lifting and carrying baby
• Sleep, activity, and nutritional considerations

• Discuss the anatomy of the tibialis posterior muscle
• Emphasize the importance of blood supply and the presence of watershed areas leading to tendon pathoanatomy
• Present the biomechanics associated with this muscles normal activity vs dysfunction
• Show clinical examples of posterior tibial tendon dysfunction
• Discuss orthotic intervention and exercises that are useful in remediation of posterior tibial tendon dysfunction